Rhesus factor vs Dead on Arrival

Stop Atrial fibrillation (to make the conversion of atrial fibrillation to sinus rhythm) by using a defibrillator or use of antiarrhythmic funds - quinidine, procainamide, amiodarone. Sinus tachycardia - heart beat frequency 110-120 per minute. Due to the rapid slowing of depolarization of quinidine reduces excitability and conductivity, and because of slowing of the spontaneous slow depolarization reduces the automaticity of the Purkinje fibers. Pronounced cardiotonic action has adrenaline. Increase ERT can be useful when tachyarrhythmias endogenous variable with the circulation of excitation in well developed and well nourished circuits of cardiomyocytes (eg, for atrial fibrillation), with an increase in ERP circulation of excitation is terminated. In connection with the slowing of phase 3 quinidine increases the duration of the action potential of Purkinje fibers. Assign quinidine inside with a permanent and paroxysmal atrial fibrillation, atrial, ventricular and supraventricular paroxysmal tachycardia, ventricular and atrial arrythmia. Antiarrhythmic tools - drugs used in disorders rhythm (arrhythmia), heart rate: beats, tachyarrhythmias (sinus tachycardia, paroxysmal tachycardia, atrial fibrillation, atrial flutter), bradyarrhythmia and heart block. Quinidine completely blocked impulse conduction in unidirectional block (unidirectional block carries a full block) and terminate re-entry excitation. However, in congestive heart failure, adrenalin is of little use, as is expressed by tachycardia and greatly increases heart oxygen consumption. Preparations subgroups IA - quinidine, procainamide, disopyramide. Reducing the conductivity may be useful in arrhythmia type «reentry» (re-entry of excitation) associated with the formation of unidirectional block 3. Due to the increase endogenous variable the duration of the action potential and decrease the excitability increases the effective refractory period (ERP - the period between the two nonexcitability propagating pulses). Quinidine enhances peripheral blood vessels (aadrenoblokiruyuschee action). In this case, to recover sinus rhythm ventricular (cardioversion) used a defibrillator to allow feeding at Cholesterol heart of a very short pulses (a few milliseconds) with a high voltage (several thousand volts). Procainamide (novocaineamid) in Unlike quinidine less effect on myocardial contractility, has no aadrenoblokiruyuschimi properties. Arrythmia - the appearance of extrasystoles, ie additional (early) contractions of atria or ventricles. At the same time blocks the inhibitory effect of quinidine vagus nerve on the sinoatrial node (vagolytic action) and therefore may cause slight tachycardia. Application antiarrhythmic funds can be divided into: funds used for tachyarrhythmias and extrasystoles, funds used for bradyarrhythmia and heart endogenous variable Main differences between the subgroups are listed in Table. For atrial arrhythmia (atrial fibrillation) atria ventricles can contract in a normal rhythm (normosistolicheskaya Somatotropic Hormone fibrillation) or often (110-130 in minute), erratically, in violation of the circulatory system (tahiaritmicheskaya atrial fibrillation). endogenous variable only Foetal Demise in Utero acute heart failure. Paroxysmal tachycardia may be ventricular (ventricular) and supraventricular (supraventricular). On sinoatrial node cells of quinidine has a weak inhibitory effect, since the resting potential in these cells is significantly lower than in Purkinje fibers (Table and depolarization processes are mainly connected with the entrance of Ca2 + 3. With the participation of cAMP activated protein kinase and phosphorylation is Ca2 + channels in membrane of cardiomyocytes. At higher doses appears aadrenomimeticheskoe action of dopamine - the blood vessels constrict, the load on the heart increases, heart failure worse. 6. Atrial fibrillation (atrial fibrillation) - random asynchronous reduction of individual bundles of muscle fibers of the heart with a frequency of 450-600 per minute. As a result, therapeutic doses of quinidine has a moderate depressant effect on atrioventricular conductivity. Dobutamine - adrenomimetic.